Effect of various nerve decompression procedures on the functions of distal limbs in streptozotocin-induced diabetic rats: Further optimism in diabetic neuropathy

Kale B., Yuksel F., Celikoz B., Sirvanci S., Ergun O., Arbak S.

PLASTIC AND RECONSTRUCTIVE SURGERY, cilt.111, sa.7, ss.2265-2272, 2003 (SCI-Expanded) identifier identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 111 Sayı: 7
  • Basım Tarihi: 2003
  • Doi Numarası: 10.1097/01.prs.0000060100.80687.d9
  • Dergi Adı: PLASTIC AND RECONSTRUCTIVE SURGERY
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.2265-2272
  • Acıbadem Mehmet Ali Aydınlar Üniversitesi Adresli: Hayır

Özet

It is known that diabetic neuropathy is the result of endoneurial edema caused by various biochemical reactions triggered by hyperglycemia. This sequence of events can cause cessation of circulation at the perineurial level, or the tough layer, which is not resilient enough to spread intraneural pressure. Internal and external limiting structures create a double crush phenomenon to the nerve structure. Decompression of the nerve trunk at separate levels is one of the adjuncts to the overall treatment plan for diabetic neuropathy.