Atıf İçin Kopyala
Koken T., Serteser M., Kahraman A., Akbulut G., Dilek O.
CLINICAL BIOCHEMISTRY, cilt.37, sa.1, ss.50-55, 2004 (SCI-Expanded)
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Yayın Türü:
Makale / Tam Makale
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Cilt numarası:
37
Sayı:
1
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Basım Tarihi:
2004
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Doi Numarası:
10.1016/j.clinbiochem.2003.10.001
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Dergi Adı:
CLINICAL BIOCHEMISTRY
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Derginin Tarandığı İndeksler:
Science Citation Index Expanded (SCI-EXPANDED), Scopus
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Sayfa Sayıları:
ss.50-55
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Anahtar Kelimeler:
ischemia-reperfusion, TNF-alpha, IL-10, anti-IL-12, oxidative stress, TUMOR-NECROSIS-FACTOR, ISCHEMIA/REPERFUSION INJURY, FACTOR-ALPHA, UP-REGULATION, IN-VIVO, ENDOTOXEMIA, FAILURE, CELLS, INVOLVEMENT, INHIBITION
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Acıbadem Mehmet Ali Aydınlar Üniversitesi Adresli:
Hayır
Özet
Objectives: Ischemia-reperfusion (I-R) injury induces production of reactive oxygen species (ROS) and some inflammatory mediators like tumor necrosis factor (TNF). We compared the effects of IL-10 and anti IL-12 antibody (Ab) on TNF-alpha production and oxidative stress markers. We also searched for which one, anti IL-12 or IL-10, is superior in the prevention of I-R induced oxidative stress.