Atıf İçin Kopyala
Demir I. E., Heinrich T., Carty D. G., Saricaoglu O. C., Klauss S., Teller S., ...Daha Fazla
EBIOMEDICINE, cilt.46, ss.431-443, 2019 (SCI-Expanded)
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Yayın Türü:
Makale / Tam Makale
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Cilt numarası:
46
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Basım Tarihi:
2019
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Doi Numarası:
10.1016/j.ebiom.2019.07.055
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Dergi Adı:
EBIOMEDICINE
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Derginin Tarandığı İndeksler:
Science Citation Index Expanded (SCI-EXPANDED), Scopus
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Sayfa Sayıları:
ss.431-443
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Anahtar Kelimeler:
Pain, Neurology of the digestive tract, nNOS, Nitrergic, Chronic pancreatitis, Pancreatic cancer, Genetically engineered mice, Atg5, NITRIC-OXIDE SYNTHASE, DUCTAL ADENOCARCINOMA, NEURONAL PLASTICITY, NEURAL PLASTICITY, SENSORY NEURONS, PROGRESSION, CELLS, INFLAMMATION, INITIATION, CANCER
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Acıbadem Mehmet Ali Aydınlar Üniversitesi Adresli:
Evet
Özet
Background: Pain due to pancreatic cancer/PCa or chronic pancreatitis/CP, is notoriously resistant to the strongest pain medications. Here, we aimed at deciphering the specific molecular mediators of pain at surgical-stage pancreatic disease and to discover novel translational targets.