Kong, B. Et Al. 2016. A subset of metastatic pancreatic ductal adenocarcinomas depends quantitatively on oncogenic Kras/Mek/Erk-induced hyperactive mTOR signalling. GUT , vol.65, no.4 , 647-657.

Kong, B., Wu, W., Cheng, T., Schlitter, A. M., Qian, C., Bruns, P., ... Jian, Z.(2016). A subset of metastatic pancreatic ductal adenocarcinomas depends quantitatively on oncogenic Kras/Mek/Erk-induced hyperactive mTOR signalling. GUT , vol.65, no.4, 647-657.

Kong, Bo Et Al. "A subset of metastatic pancreatic ductal adenocarcinomas depends quantitatively on oncogenic Kras/Mek/Erk-induced hyperactive mTOR signalling," GUT , vol.65, no.4, 647-657, 2016

Kong, Bo Et Al. "A subset of metastatic pancreatic ductal adenocarcinomas depends quantitatively on oncogenic Kras/Mek/Erk-induced hyperactive mTOR signalling." GUT , vol.65, no.4, pp.647-657, 2016

Kong, B. Et Al. (2016) . "A subset of metastatic pancreatic ductal adenocarcinomas depends quantitatively on oncogenic Kras/Mek/Erk-induced hyperactive mTOR signalling." GUT , vol.65, no.4, pp.647-657.

@article{article, author={Bo Kong Et Al. }, title={A subset of metastatic pancreatic ductal adenocarcinomas depends quantitatively on oncogenic Kras/Mek/Erk-induced hyperactive mTOR signalling}, journal={GUT}, year=2016, pages={647-657} }